Role of Arginine-Vasopressin V1a receptors for secondary brain damage following controlled cortical impact in mice

Raimund Trabold, Katrin Rauen & Nikolaus Plesnila
Objective: Brain edema formation and subsequent intracranial hypertension are well known to result in secondary brain damage and, hence, unfavorable outcome following traumatic brain injury (TBI). So far the knowledge on the molecular mechanisms of brain edema formation is still insufficient. Previously[for full text, please go to the a.m. URL]