Roles of ADMA and endothelin-1 in cerebral vasospasm after subarachnoid hemorrhage

Carla S. Jung, Bettina Lange, Klaus Zweckberger, Daniel Haux, Volker Seifert & Andreas W. Unterberg
Objective: Delayed cerebral vasospasm after aneurysmal subarachnoid hemorrhage (SAH) leading to delayed ischemic neurological deficits, may be evoked by a disturbance in vasomotor equilibrium, to which two major physiologically antagonistic compounds nitric oxide (NO) and ET-1 may contribute. Increased[for full text, please go to the a.m. URL]