Induced Hypercapnia enhances cerebral blood flow and cerebral oxygen saturation before, during and after cerebral vasospasm after aneurysmal subarachnoid hemorrhage

Christian Stetter, Thomas Westermaier, Nadine Willner, Ralf-Ingo Ernestus, Ralf Muellenbach & Ekkehard Kunze
Objective: In spite of advanced monitoring tools and pharmacological and endovascular treatment options, the occurrence of secondary ischemic deficits and infarctions after aneurysmal subarachnoid hemorrhage (SAH) is still an unsolved problem. This study was conducted to evaluate if the physiological[for full text, please go to the a.m. URL]