Inhaled nitric oxide reduces secondary brain damage after experimental traumatic brain injury

Nicole A. Terpolilli, Seong-Woong Kim, Serge C. Thal, Wolfgang M. Kuebler & Nikolaus Plesnila
Objective: Ischemia, especially pericontusional ischemia, is one of the leading causes of secondary brain damage after traumatic brain injury (TBI). So far efforts to improve cerebral blood flow after TBI were not successful. We previously demonstrated that nitric oxide (NO) applied by inhalation after[for full text, please go to the a.m. URL]