Carnosine inhibits the growth of glioblastoma cells which are not affected by inhibition of PI3K and mTOR signaling

Henry Oppermann, Helene Riedel, Lutz Schnabel, Katja Jähne, Jürgen Meixensberger & Frank Gaunitz
Objective: Signaling via receptor tyrosine kinases and the PI3K/AKT and the ERK/mTOR pathway has been considered as a possible target for the therapy of glioblastoma. As recent experiments pointed toward the possibility that the naturally occurring dipeptide carnosine may exert its anti-neoplastic[for full text, please go to the a.m. URL]
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