Pro-inflammatory TH1-response in SIRS is attenuated by cannabis (delta9-THC) supplemented opioid therapy independently of the MU-opioid receptor (µOPR) Polymorphism A118G

Lea Fritzenschaft, Knut M. Wittkowski, Weidong Du, Manfred Weiss, Thomas O. Joos, Marion E. Schneider, Peter Steffen & Stephan Paschke
Objectives: Major inflammation induced by tissue damage and the release of danger associated molecular patterns (DAMPs) conditions for immune dysfunction against nosocomial and community derived infectious agents. The inflammatory response after surgery may further influenced by perioperative pain therapy.[for full text, please go to the a.m. URL]
This data repository is not currently reporting usage information. For information on how your repository can submit usage information, please see our documentation.