Data from: Cd36-deficient congenic strains show improved glucose tolerance and distinct shifts in metabolic and transcriptomic profiles

Ondrej Šeda, Lucie Šedová, Frantisek Liška, Drahomira Křenová, Ludmila Kazdová, Johanne Tremblay, Michaela Krupková, Gilles Corbeil, Pavel Hamet & Vladimir Křen
Deficiency of fatty acid translocase Cd36 has been shown to have a major role in the pathogenesis of metabolic syndrome in the spontaneously hypertensive rat (SHR). We have tested the hypothesis that the effects of Cd36 mutation on the features of metabolic syndrome are contextually dependent on genomic background. We have derived two new congenic strains by introgression of limited chromosome 4 regions of SHR origin, both including the defective Cd36 gene, into the genetic...
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